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doi: 10.1016/j.heliyon.2024.e24302 155 ZariA

Y., Kostenko, E

Studies indicate that 50 M AS-IV significantly inhibits GSK-3 kinase activity in 2-DG-induced PC12 cells, prevents mitochondrial permeability transition pore (mPTP) opening, downregulates IRE1 and p-PERK, and thus mitigates mitochondrial damage and apoptosis under ERS (Fu et al., 2020)

No randomised controlled trials demonstrate clinically meaningful weight loss from lipotropic injections independent of lifestyle modification

The original research described it as a hepatocyte growth factor (HGF) mimetic with substantial potency in animal models of cognitive impairment