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The distribution of GSTHs is even more puzzling as from current available genomes, they are only found in S

Finally, therapeutic opportunities and ongoing preclinical/clinical studies targeting these pathways are comprehensively evaluated

Loss of HDLs anti-apoptotic capacity has been proposed to be due to increased apoCIII and possibly decreased clusterin levels on HDL (840)

While this finding is of great interest to the field of konzo as well as other neurodegenerative diseases, we cannot determine if the reduction in GPx3 is a result of disease or an inherent contributing factor for disease that is triggered by a micronutrient deficiency or underlying genetic variability