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Copper can interact with p53, playing a crucial role in regulating cell cycle progression and apoptosis ( 2+ exposure induces oxidative stress response by increasing ROS and protein carbonyl compound (PC) levels, downregulating antioxidant superoxide anion (ASA), anti-hydroxyl radical (AHR), superoxide dismutase (SOD), catalase (CAT), and glutathione peroxidase (GSH-Px) activities, and reducing glutathione (GSH) content and mRNA expression levels ( Copper ion carriers produce reactive oxygen species upon binding to copper, inducing oxidative stress, damaging DNA, and causing cell cycle arrest ( 2+ and Cu 2+ , exerting effects in various cancer cell models

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Mirhadi, A.S

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Therefore, ketone bodies have a neuroprotective effect on the central nervous system [42]