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DOX release from the micelles in the presence of 20 mM GSH was faster than in the absence of GSH due to the fracture of the disulfide bond in HA-ss-FA molecules in the reductive environment, and the released amount of DOX in PBS with 20 mM GSH was 1.4-fold more than in PBS without GSH, at 27 h

Under pathological conditions such as endotoxemia and intestinal barrier dysfunction, dysbiosis-driven metabolic reprogramming converts entero-derived metabolic signals into pro-inflammatory cytokine cascades, thereby potentiating pulmonary inflammatory microenvironment formation

J Bot (2011) 2011:19

SAR and lead optimization of (Z)-5-(4-hydroxy-3-methoxybenzylidene)-3-(2-morpholinoacetyl) thiazolidine-2, 4-dione as a potential multi-target antidiabetic agent

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