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In addition, extending the studies about their mechanisms of action for inducing allergic inflammation is necessary to improve our knowledge of allergy pathophysiology and to discover other options of allergy treatment
At the molecular level, these observations are corroborated by the disrupted regulation of proteins that promote cell proliferation, such as cyclin D1, and those that form barrier-tight junctions, including zonula occludens-1, occludin, and claudins 1, 4, and 8 (Kiuchi et al., 2009)
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