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In PD models, the use of dependent sodium channel blockers reduces glutamatergic hyperactivity and inhibits abnormal glutamate release, suggesting that amino acid metabolism disturbances may contribute to neurotoxicity and oxidative toxicity, ultimately resulting in dopaminergic neuron death [85]

For MST analysis, Syn variants, Syn WT -141C, Syn S129E -141C, and Syn 1103 -104C with a single cysteine residue near the C-terminus end were labeled using the Monolith NT Protein labeling Kit RED-MALEIMIDE (NanoTemper Technologies)

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Genipin cross-linked decellularized nucleus pulposus hydrogel-like cell delivery system induces differentiation of ADSCs and retards intervertebral disc degeneration