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P53 activation can sensitize cells to ferroptosis after ROS-induced stress by repressing the expression of GPX4 and system Xc component SLC7A11, which limits GPX4 substrate availability [61, 63,64,65,66]
Ferroptotic therapy in cancer: benefits, side effects, and risks
Its limited absorption is attributed to several factors, including interaction with P-glycoprotein (P-gp) efflux pumps, self-aggregation in the acidic environment of the gastrointestinal tract, and extensive first-pass hepatic metabolism [17,164,165]
[PMC free article] [PubMed] [Google Scholar] 107.Choi A.M., Ryter S.W., Levine B
