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Description
This leads to the production of harmful byproducts, osmotic stress, and reactive oxygen species, causing oxidative stress, DNA damage, and endoplasmic reticulum stress

Journal of Neurology, Neurosurgery & Psychiatry 87(6):670675 Sinz EH et al (1998) Quinolinic acid is increased in CSF and associated with mortality after traumatic brain injury in humans
Plant J 6:271282

This suggests that OS may broadly affect receptor function by disrupting the kinase-phosphatase equilibrium

Therapeutic potentials
