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AOD-9604 achieves selective fat metabolism by targeting beta-3 adrenergic receptors in adipose tissue without binding to growth hormone receptors that mediate IGF-1 production, glucose metabolism, and tissue growth

26 Biochemical abnormalities commonly seen in patients with ASD include impaired methylation, sulphation capacities and low glutathione (GSH) redox capacity, all of which can potentially respond to B12 supplementation

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IGF-1 LR3 remains an agonist of the IGF-1 receptor, but it has very low affinity for IGFBPs, which inhibit IGF-1, making it more potent