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glutathione arteries Repression of the antiporter SLC7A11/glutathione/glutathione peroxidase 4 axis drives ferroptosis of vascular smooth muscle cells to facilitate vascular calcification High plasma concentrations of vanin-1

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[DOI] [PubMed] [Google Scholar] 557.Centeio R., Ousingsawat J., Schreiber R., Kunzelmann K

glutathione arteries Repression of the antiporter SLC7A11/glutathione/glutathione peroxidase 4 axis drives ferroptosis of vascular smooth muscle cells to facilitate vascular calcification High plasma concentrations of vanin-1

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glutathione arteries Repression of the antiporter SLC7A11/glutathione/glutathione peroxidase 4 axis drives ferroptosis of vascular smooth muscle cells to facilitate vascular calcification High plasma concentrations of vanin-1

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glutathione arteries Repression of the antiporter SLC7A11/glutathione/glutathione peroxidase 4 axis drives ferroptosis of vascular smooth muscle cells to facilitate vascular calcification High plasma concentrations of vanin-1

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glutathione arteries Repression of the antiporter SLC7A11/glutathione/glutathione peroxidase 4 axis drives ferroptosis of vascular smooth muscle cells to facilitate vascular calcification High plasma concentrations of vanin-1

Mol Cell 2014;56:205-18

glutathione arteries Repression of the antiporter SLC7A11/glutathione/glutathione peroxidase 4 axis drives ferroptosis of vascular smooth muscle cells to facilitate vascular calcification High plasma concentrations of vanin-1

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