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These results indicate that mitochondrial dysfunction (potentially associated with reduced availability and activity of CoQ 10 ) in AD is associated with the onset of the disease rather than its progression

Once an mtDNA mutation reaches an appreciable level within cells, the percentage of mutant mtDNAs can drift by replicative segregation

It represents a fundamentally different absorption mechanism, one that bypasses the gastric destruction pathway entirely through phospholipid membrane fusion

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