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Folic acid and vitamin B12 act in synergy in the formation of DNA, and deficiencies can have serious consequences, both in performance horses with a high tissue turnover rate, and in pregnancy and growth of young foals.

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Notably, L2HG-induced ferroptosis occurs independently of impaired cystine uptake, transsulfuration pathway activity, or increased GPX4-mediated GSH utilization, revealing a non-canonical ferroptosis mechanism driven by enhanced GSH catabolism

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