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Description
Bahnam, P

Excess of glucocorticoid induces cardiac dysfunction via activating angiotensin II pathway
In this case, during morphine treatment, morphine/MOR complexes would accumulate at the plasma membrane and recruit signaling pathways involved in tolerance such as ACase superactivation and NMDA receptor regulation (Finn and Whistler, 2004)

Itching or rash
The present study hence focused on compounds, already in clinical use, with the potential to activate the Nrf2 stress response pathway
