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The knowledge on glutathione-mediated conjugation of anticancer drugs presented in this review may be noteworthy for improving cancer therapy and preventing drug resistance in cancers

Recent decades have seen a marked decrease in the molecular mechanisms involving increased reactive oxygen species overload and sperm DNA damage, as shown in the experiments by various research teams [26, 29, 30], and impaired mitochondrial function, which collectively reduce fertility potential [31]

With the discovery of metabolic reprogramming in DKD and its impact on kidney damage, there is potential for drugs targeting metabolic reprogramming to halt disease progression at a pathogenic level

These use the AdoB 12 (adenosylcobalamin) form of the vitamin