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Tatsunami R, Oba T, Takahashi K, Tampo Y (2009) Methylglyoxal causes dysfunction of thioredoxin and thioredoxin reductase in endothelial cells

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Reproducing complex, multi-component systemssuch as hybrid cell membrane-coated nanoparticles or hydrogels with precisely tuned degradation and sequential drug release profiles ( Furthermore, immunogenicity remains a critical safety concern

[DOI] [PubMed] [Google Scholar] 43.Glycine and N-acetylcysteine (GlyNAC) supplementation in older adults improves glutathione deficiency, oxidative stress, mitochondrial dysfunction, inflammation, insulin resistance, endothelial dysfunction, genotoxicity, muscle strength, and cognition: results of a pilot clinical trial

Since gamma-glutamyl transpeptidase is the only enzyme capable of starting the catabolism of GSH, GSSG, and glutathione S-conjugates, it has been proposed that it is the enzyme responsible for the regulation of glutathione movement in mammal cells [97,98]