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When activated, NLRP3 forms a high-molecular-weight intracellular complex with a number of other protein partners to regulate the maturation and secretion of pro-inflammatory cytokines such as IL-1 [10,4345]

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Mechanistically, NNMT suppression increased adipose SAM and NAD, upregulated polyamine flux enzymes (ODC, SSAT), and altered histone methylation consistent with a shift toward oxidative metabolism [2]

Delaying the inevitable: are disease modifying drugs for progressive MS worthwhile

Antioxidant therapy has demonstrated promising therapeutic effects in diseases where oxidative stress is a primary pathogenic factor and has entered clinical trials as an adjunct treatment method for various other diseases