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Description
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Additionally, many transcriptional factors and signaling proteins and other non-histone proteins also act as targets of HDAC to regulate a variety of biological functions (Song et al., 2015) indicated that the sphingosine-1-phosphate (S1P) and RhoA/ROCK1 signals may mediate cavernous fibrosis via cooperating with TGF- after CNI, which also supports the above hypothesis

Statistical analysis We analyzed data using Review Manager 5.4 software (Manager, 2020) for the statistical analyses

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have found that mitochondria play a central role in cysteine deprivation-induced ferroptosis [230]