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doi: 10.3389/fimmu.2021.665901 98 ZhouZ.WangT.JiaY.WangT.LuoE.ZhongJ.et al

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Excess Glx could drive Glu-mediated excitotoxicity, oxidative stress, and mitochondrial dysfunction, while insufficient GABAergic inhibition may exacerbate neuroinflammation, further disrupting cognitive processing, attentional control, and executive function
