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As canonical outputs of the UPRmt, mitokines represent a pivotal transition point where mitochondrial stress signaling extends beyond organelle-level homeostasis to orchestrate whole-body metabolic regulation

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The vehicle matters in dermatology

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[Abstract]2025 Oct;26(10):1660-1672

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316 Mutations in OXPHOS mtDNA lead to the inhibition of the respiratory chain, which can then increase the production of ROS and further enhance the mtDNA damage

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SahH phosphorylated by PknB c was found to be dephosphorylated by PstP c in a time-dependent manner (Fig

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