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Description
As canonical outputs of the UPRmt, mitokines represent a pivotal transition point where mitochondrial stress signaling extends beyond organelle-level homeostasis to orchestrate whole-body metabolic regulation

The vehicle matters in dermatology

[Abstract]2025 Oct;26(10):1660-1672

316 Mutations in OXPHOS mtDNA lead to the inhibition of the respiratory chain, which can then increase the production of ROS and further enhance the mtDNA damage

SahH phosphorylated by PknB c was found to be dephosphorylated by PstP c in a time-dependent manner (Fig
