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A study with pure enzyme and cell-free extracts

Although rescue of cells is partly possible by restoring anaplerosis with methyl pyruvate, failure to restore NHE activity and the cellular acidosis preclude full recovery underlining the importance of both GDH and NHE in cell survival[26]

Paul Farmer's evocative characterization of inequality as the plague of our era captures the pervasive and enduring nature of this issue (83)

Indeed, disorders of copper metabolism such as Menkes disease and Wilson disease, as well as diseases linked to dysfunction of copper-requiring enzymes, such as SOD1-linked amyotrophic lateral sclerosis, demonstrate the dramatic neurological consequences of altered copper homeostasis

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