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In people with myasthenia gravis, however, the muscle endplates have a compromised architecture that is significantly smoother and with much fewer acetylcholine receptors than their healthy counterparts

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The medication triggers the death of the inflammation-driving B-cells via two main mechanisms: antibody-dependent cellular cytotoxicity (ADCC) and complement-dependent cytotoxicity (CDC)

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10.2174/1381612043452721 104 HoustonD
