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Its modifications (Arg3 substitution and an extended N-terminus) reduce binding to IGFBPs and prolong systemic activity
Its pathophysiology involves a complex interplay of genetic, behavioral, and environmental factors, often leading to endothelial dysfunction, increased vascular resistance, and ultimately, various cardiovascular complications (Berenyiova et al
Lipids 1866 , 159030 (2021)
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doi: 10.1038/s41392-020-00261-0
