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Supplementation with NMN or overexpression of QPRT to refill the NAD + pools restored mitochondrial respiration and SIRT3 activity and normalized TCA cycle intermediates to homeostatic, anti-inflammatory levels during LPS-induced M1 macrophage polarization [30]

1) through a series of enzymatic reactions involving metabolites such as 5-hydroxytryptophan, serotonin, and N-acetylserotonin [23]

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