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In this review, we discuss how cancers obtain cysteine, how this promotes cancer cell survival, and how this may ultimately represent an exploitable metabolic vulnerability
MahajanAHerrmannMMuozLE
Any clinic that skips this step is prioritizing speed over safety
These observations revealed a previously unrecognized role of ferroptosis in ILC2 homeostasis, providing a promising therapeutic opportunity for mucosal inflammatory disorders
cerevisiae to increase its tolerance to isobutanol (Kuroda et al
