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The mechanism may be related to curculigoside significantly reducing AD-promoting factors (e.g., A1-42, p-tau) and increasing ferroptosis protective factors (e.g., GPX4, SLC7A11, GSH) in the hippocampus and cortex of AD mice, 574 suggesting that the natural compound curculigoside can be used as a promising therapeutic agent to improve AD by inhibiting ferroptosis

This process prevents cascade oxidative damage to proteins, lipids, and DNAthe building blocks of healthy cells

Although many epidemiological and genetic studies have been conducted in clinical practice, the specific molecular mechanism by which ferroptosis mediates the increased risk of DM has not been extensively studied in humans, and more studies have been carried out in animal and cell models