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CC is characterized by weight loss, primarily from muscle and body fat, resulting in muscle wasting 258

NNMT inhibition significantly increases energy expenditure, reduces body weight and white adipose mass, and improves insulin sensitivity

Even its modified version, AOD-9604, failed to produce significant weight reduction in clinical settings

Originally developed by NeuroSearch as a treatment for neurodegenerative diseases (Parkinsons and Alzheimers), tesofensine was found during clinical trials to produce significant body weight changes as a side effect, redirecting its development toward metabolic research

Mechanism of Action Inhibits Nicotinamide N-Methyltransferase (NNMT) Increases intracellular nicotinamide and NAD levels Improves mitochondrial efficiency and energy metabolism Supports lipolysis and fat oxidation Reduces adipocyte (fat cell) growth and proliferation Modulates inflammatory pathways and insulin signaling These effects may lead to enhanced fat metabolism, better blood sugar regulation, improved endurance, and a general increase in metabolic flexibility