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Description
GHK-Cu research peptide with Janoshik-verified 99.925% HPLC purity for batch PP2602-012AA, amount data, endotoxin documentation, and RUO labeling.

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Cellular communication pathways

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Mechanistically, SLC25A39 deficiency induced mitochondrial dysfunction, characterized by excessive mitochondrial reactive oxygen species (ROS), reduced membrane potential, diminished adenosine triphosphate (ATP) synthesis, aberrant mitochondrial permeability transition pore (mPTP) opening, and cytochrome c release
