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Chen Y, Tang YY, Yang L, Xiong AZ, Wang JT

Niu et al., 2012

This raises the hypothesis that similar pathways could, under specific pathological conditions, affect neurodevelopment or contribute to neurological disorders, but further studies are needed to test this connection

When viewed in this context, it led us to ask whether parkin-dependent, redox-based mechanisms could explain heretofore unresolved findings from unbiased, proteomic-based investigations of parkin-deficient mice

Group 7 received local MSC and G-CSF via Hydrogel, an intravenous injection of MSC, and an injection into the gastrocnemius muscle with MSC and G-CSF dissolved in saline