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Glucagon deficient mice exhibit perturbed zonation profiles with dampened periportal gene expression and extended glutamine synthase expression, indicating a shift toward a more pericentral like phenotype (Cheng et al., 2018)
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Exogenous copper increases the ubiquitination of GPX4 through direct binding to the cysteine residues C107 and C148 of the GPX4 protein, promoting the formation of GPX4 aggregates
