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Instead, astrocytes may mainly participate in -amyloid clearance through various mechanisms, such as producing -amyloid-degrading proteases, extracellular APOE, ApoJ/Clusterin, 1-antichymotrypsin (ACT) and 2-macroglobulin (2-M

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although, IL-1 and IL-18 protein levels in scopolamine-treated mice were significantly reduced as compared to those in the surgery group (Fig

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