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Journal of cachexia, sarcopenia and muscle, 1(1), 23-33

The delay in 53BP1 recruitment to DSBs in these cells and the accumulation of irreparable damage may be a potent physiological genotoxic stress in individuals with HGPS

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The product of this reaction, phosphatidyl ethanol, is poorly metabolized and may accumulate to detectable levels following chronic consumption of large amounts of alcohol, but its effects on the cell remain to be established