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Thats because neither its therapeutic potential nor its safety has been tested on people

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Additional research has explored its influence on regulatory protein networks, transcription factor activity, and downstream signal transduction systems associated with telomere biology and gene regulation

[PubMed] [Google Scholar] 56.Bakhtogarimov I.R., Kudryavtseva A.V., Krasnov G.S., Gladysh N.S., Volodin V.V., Kudryavtsev A.A., Bulavkina E.V., Goncharova M.A., Ledyaeva V.S., Pastukhov I.S., et al

They: Produce collagen Organize extracellular matrix Restore tensile structure In experimental tendon transection models, BPC-157 has been associated with: Increased fibroblast density Enhanced migration into injury site Accelerated granulation tissue formation Proposed signalling involvement includes: FAK (Focal Adhesion Kinase) activation ERK1/2 signalling Src pathway modulation FAK signalling is particularly important because it regulates: Cell adhesion Cytoskeletal reorganization Migration along collagen scaffolds Tendon strength depends not just on collagen quantity but alignment