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(Wan et al., 2016) found that the system could restore the NP phenotype following dedifferentiation through upregulating NP-specific genes ( KRT-8, KRT-18, FOXF-1 ) and the SAPH stimulated time-dependent increases in aggrecan and type II collagen deposition (Figure 7)

10.1016/j.neuron.2011.12.031 86 PanenkaW

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In this review, we will examine the potential of mitochondrial dysfunction in PBMCs as a potential diagnosis/prognosis marker for MASLD

Inhibition of Mrp2- and Ycf1p-mediated transport by reducing agents: evidence for GSH transport on rat Mrp2
