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Oral glucose tolerance tests performed in clinical trials showed no significant changes in glucose handling, and fasting insulin levels remained stable throughout treatment periods

found that emodin can inhibit the growth of HepG2 cells in a dose - and time-dependent manner induce cell S phase and G2/M phase stagnation promote apoptosis of HepG2 cells and thus control the migration and invasion of HepG2 cells

Colony formation assay By cell counting, 1000 cells were grown in culture in six-well culture plates

Table 1 displays the primer sequences of target genes using the housekeeping gene -actin as internal control along with the amplified PCR product sizes

Hall A (1999) The role of glutathione in the regulation of apoptosis