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This explicit requirement for lipid peroxidation is a core feature of ferroptosis that distinguishes it from other forms of cell death: ferroptosis inhibitors cannot prevent other forms of cell death (Yang et al., 2014), and conversely classic inhibitors of necrosis, apoptosis, and autophagy do not modulate ferroptosis ( Figure 1 )

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References 113 and 114 highlighted the anti-tumoural benefits of co-targeting pericytes and ECs in mouse cancer models, paving the way to the clinical testing of non-selective VEGFR and PDGFR inhibitors in PNETs and other cancer types

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