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On the other hand, increased catecholamines due to MAO inhibition have been shown to reduce lymphocyte proliferation in a process mostly dependent on 2 adrenergic receptors (ARs) [43]

Gliotoxin can target the 20S proteasome, which is responsible for the degradation of the IK-B protein that specifically binds to NF-B

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Pharmacological stabilization of hypoxia-inducible factor 1- dampens the interferon response and promotes glycolysis in Aicardi-Goutires syndrome
