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In vivo studies in different acute liver failure (ALF) animal models have shown the existence of cell death by necrosis resulted in rapid IL-1 precursor release and upregulation of IL-1 and IL-18, leading to tissue injury (Sakurai et al., 2008

These organisms serve as prey for higher-level organisms, which selectively accumulate and metabolize carotenoids, including precursor forms and AX

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