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NTZ-elicited hepatoprotective effects are suggested to be mediated via modulation of PERK/CHOP10 signaling along with attenuated proapoptotic oxidative stress and proinflammatory signaling

This enzyme generates 5-oxoproline and releases the amino acid or peptide that is bound to glutamate

In particular, exposure to doxorubicin augmented mitochondrial dysfunction in GPx1-deficient cardiomyocytes compared to normal cardiomyocytes, uncoupling electron transfer and oxidative phosphorylation

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Studies have revealed that antitumor medications heavily rely on GPX4