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doi: 10.2478/enr-20220031 87 EnglandJDGronsethGSFranklinGMillerRGAsburyAKCarterGTet al

10.1186/scrt506 [DOI] [PMC free article] [PubMed] [Google Scholar] Murillo G., Peng X

It is therefore suggesting that the slight change of relaxation observed in the aorta without endothelium ex vivo might be due to some mechanism unrelated to the direct nitric oxide stimulation which needs further investigation

doi: 10.1038/s41556-024-01360-8 7 DixonSJLembergKMLamprechtMRSkoutaRZaitsevEMGleasonCEet al

Recent findings suggest that many of the memory enhancing effects originally attributed to AngII acting at the AT1 receptor subtype were due to the conversion of AngII to angiotensin III (AngIII) and then to the hexapeptide angiotensin IV (Ang IV) and it were AngIV activation of the AT4 receptor subtype that facilitated memory consolidation and retrieval [62-64]
