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The purpose of this review is threefold: first, to describe the pathophysiological mechanisms by which thyroid dysfunction affects the brain

[145], and MT-1A and MT-2A polymorphisms were associated with T2DM and coronary artery disease in the study of a Bulgarian cohort by Kozarova et al

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The interactions of MT with other hormones, as well as the mediating mechanism that enhance plant tolerance, are still unclear

An internal laqI q gene helps to maintain tight control over expression of the insert by binding to the tac promoter until induction with IPTG