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135 Dihydroartemisinin can significantly inhibit the viability of leukemia cells, causing cell cycle G0/G1 phase arrest and ferroptosis, and the mechanism may be related to inducing autophagy and accelerating ferritin degradation by regulating AMP-activated kinase (AMPK)/mTOR/p70S6k signaling pathway
L.ConradD.et al (1999)
Bi, J.H
Beyond this window, however, appropriate movement promotes healing by increasing blood flow, preventing muscle atrophy, and maintaining neuromuscular connections
In general, the active sites of DNA glycosylases are small to accommodate large bulky base modifications
