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A.et al (2013)

In eukaryotes, TML is the product of lysosomal or proteasomal degradation of proteins such as calmodulin, myosin, actin, cytochrome c, and histones, which contain N -methylated lysine residues from post-translational modification (Strijbis et al., 2010)

Improvements in electrophysiological parameters were also observed with ALCAR treatment (93)

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