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Activation of Akt1 is usually cytoprotective, such as its function during endothelial cell hypoxia (Somanath, Razorenova, Chen, & Byzova, 2006), and Akt1 has been extensively studied and is considered to be neuroprotective in stroke (Zhao, Sapolsky, & Steinberg, 2006), with Akt1 and Akt3 proteins degrading as early as 1 hr after stroke (Xie et al., 2013)

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is enacted, it does not necessarily mean the drugs are safe or effective

[Google Scholar] 5.Aksoy S, Szumlanski CL, Weinshilboum RM