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The amyloid cascade hypothesis which is based on the molecular defects observed in autosomal-dominant EOAD has been severely criticized due to the unsolved paradox: Why do A aggregate into fibrils?, but it is clear that the A sequence, A concentration, and conditions that destabilize A are important factors in aggregation [83, 84]

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Premature aging and metabolic diseases: the impact of telomere attrition

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300 Diabetes Insulin resistance and high blood glucose levels increase oxidative stress, trigger vascular inflammation and endothelial dysfunction, activate platelets and pro-coagulation proteins, and inhibit fibrinolysis