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5759 Increasing data indicates that the maintenance of high NAD+ is critical to SIRT1-mediated activation of autophagy pathways and the clearance of damaged cellular proteins

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64 As a result of fibrin formation, secondary fibrinolysis occurs and this generates D-dimer, with levels paralleling the degree of inflammation

Additionally, obesity and IR both contribute to -cell dysfunction and apoptosis, mechanistically explained by glucotoxicity and increased immunogenicity (the accelerator hypothesis) or by -cell exhaustion due to increased metabolic demand (the overload hypothesis) [23]