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Porter A, Godbold JA, Lewis CN, Savage G, Solan M, Galloway TS
In summary, a defect in the methionine biosynthesis pathway at low pH renders the metZ strain metabolically less active, thereby inhibiting its growth at acidic pH
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Likely, this may be by rapidly activating alternative bypassing pathways
doi: 10.1023/A:1010255230338
Lysine residues in protein can be methylated by lysine methyltransferases (PKMTs) with S-adenosyl-L-methionine (AdoMet) functioning as the primary methyl group donor
