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[102] who showed that the incorporation of 14 C into lipids was highest at 1 hour after injection of [1- 14 C]acetyl-L-carnitine into adult rat brain, and labeling was decreased at 3, 6 and 22 h after injection, suggesting that turnover of lipids containing carbons from ALCAR and reutilization of the carbons from ALCAR occurs in brain [102]

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These organs are known to have high concentrations of mitochondria

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10.1007/s10753-015-0261-7 53

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Carcinogenic properties of proteins with pro-inflammatory activity from streptococcus infantarius (formerly s.bovis)

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Furthermore, C18:1-carnitine has been demonstrated to function as an efficacious noncompetitive inhibitor of GlyT2, which has the potential to serve as an analgesic by regulating the transport of glycine in the central nervous system (CNS) and influencing the concentration of this pivotal inhibitory neurotransmitter [112]

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