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Contextually, all attributes of mitochondrial patho-/physiology are instrumental for predictive medical approach and cost-effective treatments tailored to individualized patient profiles in primary (to protect vulnerable individuals again the health-to-disease transition) and secondary (to protect affected individuals again disease progression) care

Together, these findings suggest that SFP-1 acts as a key regulator of fat depletion to facilitate resource allocation after mating

Caveolin-1 ( CAV1 gene) is ubiquitously expressed, but shows high expression in adipocytes, fibroblasts, pneumocytes, neurons, glia, endothelial and epithelial cells [42]

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