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Mechanistically, selenium deficiency may exacerbate thyroid-related neurological dysfunction through three pathways: impaired deiodinase activity, weakened antioxidant defense, and disrupted selenium transport (134136)

Presynaptic deficits in a number of brain illnesses may result from glutamate transporter-1 (GLT-1) dysfunction because the presynaptic membrane is particularly susceptible to it

While elevated liver enzymes dont always mean liver disease, they are a helpful signal that the liver may need extra attention and support

water retention anywhere in the body

Organ-specific toxicity is another critical area of concern