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We discovered that Diphenyleneiodonium chloride (DPI), an inhibitor of flavoprotein enzymes and mitochondrial complex I, synergizes with mitoquinone mesylate (MitoQ), a mitochondria-targeted antioxidant in 2D and 3D in vitro models of NB

Genetic mutation or deficiency of PARK7/DJ-1 disrupts dopamine metabolism, induces ROS accumulation, and impairs mitochondrial function in neuronal cells

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Progression of HNSCC from hyperplasia to dysplasia is marked by the loss of 3p21 and 17p13

This gene modulation effect extends to pathways related to wound response, growth factors, and anti-fibrotic mechanisms, making it of growing interest in regenerative biology studies